QuestionSBA
QuestionChoice
Digoxin toxicity typically presents with hyperkalaemia and bradycardia with prolonged PR and QRS complexes. The most common ocular manifestation of dioxin toxicity is yellow/green tinted vision. Treatment of digoxin toxicity is with "digibind."
10026926
a
Digoxin toxicity
QuestionChoice
Beta blocker toxicity may produce bradycardia. However, it is also likely to produce hypotension, hypothermia and hypoglycaemia. It would not cause yellow tinted vision
10026928
c
Beta blocker toxicity
QuestionChoice
Rivaroxaban toxicity could result in a haemorrhagic stroke which could lead to ocular manifestations like yellow-tinted vision. However, it is unlikely to produce hyperkalaemia seen is this patient
10026929
d
Haemorrhagic stroke secondary to rivaroxaban toxicity
QuestionChoice
Early onset dementia may explain her period of confusion, however, it would not explain her hyperkalaemia, bradycardia or yellow tinted vision
10026927
b
Early onset dementia
QuestionChoice
A urinary tract infection may produce the symptoms of confusion seen in this patient. However, a UTI is unlikely to produce the bradycardia, hyperkalaemia nor yellow tinted vision that this patient is experiencing
10026930
e
Urinary tract infection
Concept
Chapter
2693
4552
Digoxin
Topic
169
Cardiovascular pharmacology
17783
An 88-year-old woman with a history of atrial fibrillation and osteoarthritis presents to A&E with her son. He says that she has been complaining of her heart "feeling funny" and has become very confused since breakfast. Additionally, she has become distressed and is complaining of "yellow vision". Her regular medications include digoxin, rivaroxaban, bisoprolol and paracetamol.

Her bloods show raised serum potassium levels and her ECG shows bradycardia with prolonged PR intervals and QRS complexes.

What is the single most likely cause of her presentation?
a