QuestionSBA
QuestionChoice
Inhibiting leukotriene receptors is not the primary mechanism of action of NSAIDs. Leukotrienes are primarily targeted by leukotriene modifiers, a different class of anti-inflammatory drugs.
10024054
e
Inhibition of leukotriene receptors
QuestionChoice
This is not the primary mechanism of action of NSAIDs. Inhibiting 5-lipoxygenase is associated with another class of anti-inflammatory drugs called leukotriene modifiers, not NSAIDs.
10024051
b
Inhibition of 5-lipoxygenase
QuestionChoice
This is an effect of NSAIDs, but the primary mechanism is the inhibition of cyclooxygenases (COX). NSAIDs can inhibit the production of prostacyclin (a vasodilator and platelet aggregation inhibitor) as a result of COX inhibition.
10024052
c
Inhibition of prostacyclin synthesis
QuestionChoice
NSAIDs do not activate COX-2; they inhibit it. COX-2 is an enzyme involved in the production of inflammatory prostaglandins. Inhibition of COX-2 is one of the key mechanisms of NSAIDs.
10024053
d
Activation of COX-2
QuestionChoice
NSAIDs work by inhibiting COX enzymes, particularly COX-1 and COX-2. This inhibition reduces the production of prostaglandins, which are involved in inflammation, pain, and fever.
10024050
a
Inhibition of cyclooxygenases (COX)
Concept
Chapter
2693
5725
Anti-Inflammatory Drugs
Topic
286
Infection/ Inflammation
17208
Which of the following describes the mechanism of action of an NSAID?
a