QuestionSBA
QuestionChoice
Inhalation of tobacco smoke activates **type II pneumocytes** and results in inflammation and the release of proteases
10026774
e
Inhalation of tobacco smoke leads to the activation of type I pneumocytes and the release of collagenase and elastase leading to the breakdown of alveolar walls
QuestionChoice
This describes chronic bronchitis.
10026771
b
Tobacco smoke leads to the hypertrophy and hyperplasia of goblet cells and mucinous glands
QuestionChoice
Alpha-1-anti-trypsin protects against the action of proteases. Deficiency results in panacinar emphysema
10026773
d
Release of alpha-1-anti-trypsin, leukotriene B4 and elastase leads to the breakdown of alveolar walls
QuestionChoice
This describes asthma.
10026772
c
Inhalation of tobacco smoke leads to bronchial hyperresponsiveness and intermittent airway obstruction
QuestionChoice
Inhalation of tobacco smoke activates type II pneumocytes and leads to inflammation and the release of proteases which breakdown alveolar walls
10026770
a
Release of leukotriene B4, collagenase and elastase leads to the breakdown of alveolar walls
Concept
Chapter
2693
3902
COPD
Topic
132
Respiratory
17752
A 64-year-old long-term smoker is diagnosed with COPD after years of suffering from exertional shortness of breath and a productive cough with sticky mucus.

Which of the following describes a pathological process involved in chronic emphysema?
a